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Combined exposure to even low doses of alcohol with THC, HU-210, or CP 55,940 caused a greater incidence of birth defects, particularly of the eyes, than did either treatment alone. Consistent with the hypothesis that these defects are caused by deficient Shh, we found that CBs reduced Shh signaling by inhibiting Smoothened (Smo), while Shh mRNA or a CB1 receptor antagonist attenuated CB-induced birth defects. Proximity ligation experiments identified novel CB1-Smo heteromers, suggesting allosteric CB1-Smo interactions. In addition to raising concerns about the safety of cannabinoid and alcohol exposure during early embryonic development, this study establishes a novel link between two distinct signaling pathways and has widespread implications for development, as well as diseases such as addiction and cancer.

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International Academy on the Science and Impact of Cannabis

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SAM

 

Frequently Asked Questions of Why We Are Opposed to Weed!

Get ya head straight!

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Marijuana Victims' Association
Windsor Morning

Busting the Myth
that Cannabis Doesn't Kill

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Dr Kevin Sabet, C.E.O of Smart Approaches to Marijuana: Presents at the 20th NADCP Annual Conference

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The Marijuana-Opioid Connection

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